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Cammack N

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Publications:

Title Authors Year Journal
Activity of the isolated HIV RNase H domain and specific inhibition by N-hydroxyimides. Cammack N 2004 Biochem Biophys Res Commun
Two-metal ion mechanism of RNA cleavage by HIV RNase H and mechanism-based design of selective HIV RNase H inhibitors. Cammack N 2003 Nucleic acids research
Resistance to (-)-2',3'-dideoxy-3'-thiacytidine (3TC) in HIV-1 isolated from paediatric patients. Cammack N 1996 Antiviral therapy
Development of HIV-1 resistance to (-)2'-deoxy-3'-thiacytidine in patients with AIDS or advanced AIDS-related complex. Cammack N 1995 AIDS (London, England)
The same mutation that encodes low-level human immunodeficiency virus type 1 resistance to 2',3'-dideoxyinosine and 2',3'-dideoxycytidine confers high-level resistance to the (-) enantiomer of 2',3'-dideoxy-3'-thiacytidine. Cammack N 1993 Antimicrob Agents Chemother
High-level resistance to (-) enantiomeric 2'-deoxy-3'-thiacytidine in vitro is due to one amino acid substitution in the catalytic site of human immunodeficiency virus type 1 reverse transcriptase. Cammack N 1993 Antimicrobial agents and chemotherapy
(-)-2'-deoxy-3'-thiacytidine is a potent, highly selective inhibitor of human immunodeficiency virus type 1 and type 2 replication in vitro. Cammack N 1992 Antimicrob Agents Chemother
The separated enantiomers of 2'-deoxy-3'-thiacytidine (BCH 189) both inhibit human immunodeficiency virus replication in vitro. Cammack N 1992 Antimicrob Agents Chemother
Single mutations at many sites within the DNA polymerase locus of herpes simplex viruses can confer hypersensitivity to aphidicolin and resistance to phosphonoacetic acid. Cammack N 1984 The Journal of general virology
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