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Oliveira M

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Publications:

Title Authors Year Journal
Antiviral activity and resistance profile of phosphazid--a novel prodrug of AZT. Oliveira M Nucleosides & nucleotides
The preferential selection of K65R in HIV-1 subtype C is attenuated by nucleotide polymorphisms at thymidine analogue mutation sites. Oliveira M 2013 The Journal of antimicrobial chemotherapy
Basis for the Early and Preferential Selection of the E138K Substitution in HIV-1 Reverse Transcriptase with Etravirine. Oliveira M 2013 Antimicrobial agents and chemotherapy
The Effect of Mutations at Position E138 in HIV-1 Reverse Transcriptase and their Interactions with the M184I Mutation in Defining Patterns of Resistance to the Non-Nucleoside Reverse Transcriptase Inhibitors Rilpivirine and Etravirine. Oliveira M 2013 Antimicrobial agents and chemotherapy
Maraviroc and other HIV-1 entry inhibitors exhibit a class-specific redistribution effect that results in increased extracellular viral load. Oliveira M 2012 Antimicrobial agents and chemotherapy
Transmission dynamics of the M184V drug resistance mutation in primary HIV infection. Oliveira M 2011 The Journal of antimicrobial chemotherapy
A template-dependent dislocation mechanism potentiates K65R reverse transcriptase mutation development in subtype C variants of HIV-1. Oliveira M 2011 PloS one
Characterization of the E138K resistance mutation in HIV-1 reverse transcriptase conferring susceptibility to etravirine in B and non-B HIV-1 subtypes. Oliveira M 2011 Antimicrobial agents and chemotherapy
Compensation by the E138K mutation in HIV-1 reverse transcriptase for deficits in viral replication capacity and enzyme processivity associated with the M184I/V mutations. Oliveira M 2011 Journal of virology
Differential impact of the HIV-1 non-nucleoside reverse transcriptase inhibitor mutations K103N and M230L on viral replication and enzyme function. Oliveira M 2010 The Journal of antimicrobial chemotherapy
Comparative biochemical analysis of recombinant reverse transcriptase enzymes of HIV-1 subtype B and subtype C. Oliveira M 2010 Retrovirology
Human immunodeficiency virus type 1 recombinant reverse transcriptase enzymes containing the G190A and Y181C resistance mutations remain sensitive to etravirine. Oliveira M 2009 Antimicrobial agents and chemotherapy
Apricitabine does not select additional drug resistance mutations in tissue culture in human immunodeficiency virus type 1 variants containing K65R, M184V, or M184V plus thymidine analogue mutations. Oliveira M 2009 Antimicrobial agents and chemotherapy
Template usage is responsible for the preferential acquisition of the K65R reverse transcriptase mutation in subtype C variants of human immunodeficiency virus type 1. Oliveira M 2009 Journal of virology
Signature nucleotide polymorphisms at positions 64 and 65 in reverse transcriptase favor the selection of the K65R resistance mutation in HIV-1 subtype C. Oliveira M 2009 The Journal of infectious diseases
Transmission networks of drug resistance acquired in primary/early stage HIV infection. Oliveira M 2008 AIDS (London, England)
Near full-length genomic analysis of a novel subtype A1/C recombinant HIV type 1 isolate from Canada. Oliveira M 2008 AIDS research and human retroviruses
Variations in reverse transcriptase and RNase H domain mutations in human immunodeficiency virus type 1 clinical isolates are associated with divergent phenotypic resistance to zidovudine. Oliveira M 2007 Antimicrobial agents and chemotherapy
Diminished efficiency of HIV-1 reverse transcriptase containing the K65R and M184V drug resistance mutations. Oliveira M 2007 AIDS (London, England)
High prevalence of the K65R mutation in human immunodeficiency virus type 1 subtype C isolates from infected patients in Botswana treated with didanosine-based regimens. Oliveira M 2006 Antimicrobial agents and chemotherapy
HIV-1 subtype C viruses rapidly develop K65R resistance to tenofovir in cell culture. Oliveira M 2006 AIDS (London, England)
Differential maintenance of the M184V substitution in the reverse transcriptase of human immunodeficiency virus type 1 by various nucleoside antiretroviral agents in tissue culture. Oliveira M 2004 Antimicrobial agents and chemotherapy
Persistence of multidrug-resistant HIV-1 in primary infection leading to superinfection. Oliveira M 2004 AIDS (London, England)
Lamivudine can exert a modest antiviral effect against human immunodeficiency virus type 1 containing the M184V mutation. Oliveira M 2003 Antimicrobial agents and chemotherapy
The M184V substitution in human immunodeficiency virus type 1 reverse transcriptase delays the development of resistance to amprenavir and efavirenz in subtype B and C clinical isolates. Oliveira M 2003 Antimicrobial agents and chemotherapy
A V106M mutation in HIV-1 clade C viruses exposed to efavirenz confers cross-resistance to non-nucleoside reverse transcriptase inhibitors. Oliveira M 2003 AIDS (London, England)
Pressure of zidovudine accelerates the reversion of lamivudine resistance-conferring M184V mutation in the reverse transcriptase of human immunodeficiency virus type 1. Oliveira M 2002 Antimicrobial agents and chemotherapy
Polymorphisms of cytotoxic T-lymphocyte (CTL) and T-helper epitopes within reverse transcriptase (RT) of HIV-1 subtype C from Ethiopia and Botswana following selection of antiretroviral drug resistance. Oliveira M 2002 Antiviral research
Co-receptor usage and HIV-1 intra-clade C polymorphisms in the protease and reverse transcriptase genes of HIV-1 isolates from Ethiopia and Botswana. Oliveira M 2002 Antiviral therapy
The M184V mutation in reverse transcriptase can delay reversion of attenuated variants of simian immunodeficiency virus. Oliveira M 2002 Journal of virology
Genetic divergence of human immunodeficiency virus type 1 Ethiopian clade C reverse transcriptase (RT) and rapid development of resistance against nonnucleoside inhibitors of RT. Oliveira M 2002 Antimicrobial agents and chemotherapy
Persistence and fitness of multidrug-resistant human immunodeficiency virus type 1 acquired in primary infection. Oliveira M 2002 Journal of virology
Selection of resistance-conferring mutations in HIV-1 by the nucleoside reverse transcriptase inhibitors (+/-)dOTC and (+/-)dOTFC. Oliveira M 2000 Antiviral chemistry & chemotherapy

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